Displaying articles by keyword ETAR

The paper presents a brief review on how some concepts on the organism general adaptational reactions developed, including their role…

Consideration is being given herein to some mechanisms of high heart rate variability (high HRV), which cannot be attributed to…

Conventional methods for arterial pressure (AP) measurement are not capable of identifying it in each cardiac cycle. Besides, they ignore…

This paper deals with solving of a century-old paradox of linear stability for the Hagen-Poiseuille flow. A new mechanism of…

Recipients of ventricular assist devices (VADR) have a higher incidence to develop antibodies (Abs) against human leukocyte antigens (HLA). Non-HLA antibodies like major histocompatibility complex class I-related chain A (MICA) and autoantibodies against angiotensin type 1 receptor (AT1R) and endothelin receptor A (ETAR) are also implicated in the pathogenesis of acute rejection and allograft vasculopathy. We monitored non-HLA- and HLA-Abs in VADR up to one year after implantation.

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